From MIT homeopathic point of view, prostaglandins potentized above 12c will be containing molecular imprints of functional groups prosatglandin molecules. These molecular imprints can act as artificial binding pockets for prosatgandins when applied as therapeutic agents. By inhibiting the prostaglandins, these molecular imprints can work as excellent anti inflammatory, analgesic and antipyretic. It can also be helpful in the treatment of false labor pains, hypertension, hyperacidity etc also.
Prostaglandins are a group of physiologically active lipid compounds having diverse hormone-like effects in animals. They are derived enzymatically from fatty acids. Every prostaglandin contains 20 carbon atoms, including a 5-carbon ring. They are synthesized in the cells of their target tissues and act near their points of synthesis, which classifies them as autocrine or paracrine signaling molecules.
Prostaglandins are synthesized from arachidonic acid, a 20-carbon unsaturated fatty acid. The enzyme phospholipase A2 releases arachidonic acid from membrane phospholipids, which is then converted to prostaglandin H2 (PGH2) by the action of cyclooxygenase (COX) enzymes. PGH2 serves as a precursor for other prostaglandins, as well as thromboxanes and prostacyclin.
Prostaglandins are classified into different series (e.g., PGE, PGF) based on their chemical structure, specifically the functional groups on their 5-carbon ring. The number following the letters (e.g., PGE2, PGF2α) indicates the number of double bonds outside the ring.
Prostaglandins have a wide range of physiological functions. They mediate inflammatory responses, promoting fever, swelling, and pain as part of the body’s defense mechanism. Prostaglandins protect the stomach lining by stimulating the secretion of mucus and bicarbonate, thereby inhibiting acid production. They are involved in ovulation, the menstrual cycle, and inducing labor by ripening the cervix and causing uterine contractions. Certain prostaglandins dilate blood vessels, contributing to blood flow regulation and blood pressure. They modulate synaptic transmission and are involved in the pain sensation. They play a role in the regulation of renal blood flow and electrolyte balance.
NSAIDs, such as ibuprofen and aspirin, inhibit cyclooxygenase (COX) enzymes, reducing the synthesis of prostaglandins and thus their inflammatory effects. There are two main types of COX enzymes: COX-1 and COX-2. COX-1 inhibitors can lead to gastrointestinal side effects, while selective COX-2 inhibitors aim to reduce these risks.
Prostaglandins are crucial in a wide range of physiological and pathophysiological processes. Their diverse roles and mechanisms of action make them significant both for understanding human biology and for therapeutic interventions. Advances in understanding their biosynthesis and function have led to the development of drugs that mimic or inhibit their action, providing critical treatments for various conditions.
From MIT homeopathic point of view, prostaglandins potentized above 12c will be containing molecular imprints of functional groups prosatglandin molecules. These molecular imprints can act as artificial binding pockets for prosatgandins when applied as therapeutic agents. By inhibiting the prostaglandins, these molecular imprints can work as excellent anti inflammatory, analgesic and antipyretic. It can also be helpful in the treatment of false labor pains, hypertension, hyperacidity etc also.
REDEFINING HOMEOPATHY 